MEDI 363 |
| Signaling through the insulin-like growth factor I (IGF-1R) pathway mediates events which promote the malignant phenotype such as mitogenisis (via stimulation of signaling cascades through Ras/Raf/MARK kinase) and cell survival (via activation of the PI3K/Akt/mTor kinase pathway). Epidemiological studies have shown that elevated IGF-1 levels correlate with increased risk of developing colon, breast, prostate, and lung tumors. Previously we reported BMS-536924 as a new small molecule kinase inhibitor of IGF-1R with robust efficacy in IGF-Sal tumor model. As part of our continuous efforts toward the identification of IGF-1 receptor kinase inhibitors with improved enzyme and cell potency, we have recently identified a novel series of 3-[(6-piperidine-4-yl)-4-methyl-1H-benzimidazol-2-yl]-1H-pyridin-2-one derivatives. The synthesis and detailed SAR of this series as IGF-1 receptor kinase inhibitors are presented. |
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Poster Session
7:00 PM-9:00 PM, Wednesday, August 22, 2007 BCEC -- Exhibit Hall - B2, Poster
Division of Medicinal Chemistry |